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Leukemia Research
Volume 30, Issue 6
, Pages 707-712
, June 2006
Clinical effects of oral green tea extracts in four patients with low grade B-cell malignancies
References
- . Inhibition of carcinogenesis by tea. Annu Rev Pharmacol Toxicol. 2002;42:25–54
- . VEGF receptor phosphorlyation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia. Blood. 2004;104:788–794
- Expression of apoptotis-regulating proteins in chronic lymphocytic leukemia: correlations with in vitro and in vivo chemoresponses. Blood. 1998;91:3379–3389
- . Mcl-1 and Bcl-2/Bax ratio are associated with treatment response but not with Rai stage in B-cell chronic lymphocytic leukemia. Am J Hematol. 2004;75:22–33
- . Inhibition of carcinogenesis by tea. Nature. 1997;389:134–135
- . Angiogenesis inhibited by drinking tea. Nature. 1999;398:381
- . Blood and urine levels of tea catechins after ingestion of different amounts of green tea by human volunteers. Cancer Epidemiol Biomarkers Prev. 1998;7:351–354
- . The intermediate filament protein vimentin is a new target for EGCG. J Biol Chem. 2005;280:16882–16890
- . Cancer chemoprevention by tea polyphenols through modulating signal transduction pathways. Arch Pharm Res. 2002;25:561–575
- National Cancer Institute-Sponsored Working Group Guidelines for Chronic Lymphocytic Leukemia: revised guidelines for diagnosis and treatment. Blood. 1996;87:4990–4997
- News TESC. Green tea confuses cancer cells. CBS newscom; 2004. http://www.cbsnews.com/stories/2004/04/01/earlvshow/contributors/emilysenav/main609880.shtml.
- World Health Organization classification of neoplastic diseases of the hematopoietic and lymphoid tissues: report of the Clinical Advisory Committee meeting—Airlie House, Virginia, November 1997. J Clin Oncol. 1999;17:3835–3849
- Report of an international workshop to standardize response criteria for non-Hodgkin's lymphomas. NCI Sponsored International Working Group. J Clin Oncol. 1999;17:1244
- . Green tea catechins inhibit vascular endothelial growth factor receptor phosphorylation. Cancer Res. 2002;62:381–385
- EGCG, a major component of green tea, inhibits tumor growth by inhibiting VEGF induction in human colon carcinoma cells. Br J Cancer. 2001;84:844–850
- . Green tea constituent epigallocatechin-3-gallate and induction of apoptosis and cell cycle arrest in human carcinoma cells. J Natl Cancer Inst. 1997;89:1881–1886
- Blocking telomerase by dietary polyphenols is a major mechanism for limiting the growth of human cancer cells in vitro and in vivo. Cancer Res. 2003;63:824–830
- . Telomerase inhibition, telomere shortening, and senescence of cancer cells by tea catechins. Biochem Biophys Res Commun. 1998;249:391–396
- . The antifolate activity of tea catechins. Cancer Res. 2005;65:2059–2064
- . Epigallocatechin gallate causes oxidative damage to isolated and cellular DNA. Biochem Pharmacol. 2003;66:1769–1778
- . Prooxidant property of green tea polyphenols epicatechin and epigallocatechin-3-gallate: implications for anti-cancer properties. Toxicol In Vitro. 2004;18:555–561
- . Generation of hydrogen peroxide primarily contributes to the induction of FE(II) dependent apoptosis in Jurkat cells by epigallocatechin gallate. Carcinogenesis. 2004;25:1567–1574
- . Green tea component, catechin, induces apoptosis of human malignant B cells via production of reactive oxygen species. Clin Cancer Res. 2005;11:6040–6049
- Specific killing of multiple myeloma cancer cells by epigallocatechin-3-gallate extracted from green tea. Blood. 2004;104:2461;[abstract]
- . DNA degradation by water extract of green tea in the presence of copper ions: implications for anticancer properties. Phytother Res. 2003;17:358–363
- . Induction of poly(ADP-ribosyl)ation and DNA damage in human peripheral lymphocytes after treatment with (−)-epigallocatechin-gallate. Mutat Res. 2003;534:77–84
- . DNA cleavage activities of (−)-epigallocatechin, (−)-epicatechin, (+)-catechin, and (−)-epigallocatechin gallate with various kinds of metal ions. Biosci Biotechnol Biochem. 1999;63:1654–1656
- . Antioxidant, gallic acid, induces apoptosis in HL-60RG cells. Biochem Biophys Res Commun. 1994;204:898–904
- . Reactivities of flavonoids with different hydroxyl substituents for the cleavage of DNA in the presence of Cu(II). Phytother Res. 1999;13:609–612
- . Putative mechanism for anticancer and apoptosis-inducing properties of plant-derived polyphenolic compounds. IUBMB Life. 2000;50:167–171
- . Cancer prevention by tea polyphenols is linked to their direct inhibition of antiapoptotic Bcl-2-family proteins. Cancer Res. 2003;63:8118–8121
- Catechin, a green tea component, rapidly induces apoptosis of myeloid leukemic cells via modulation of reactive oxygen species production in vitro and inhibits tumor growth in vivo. Haematologica. 2005;90:317–325
- . Chemoprevention of human prostate cancer by oral administration of green tea catechins (GTCs) in high grade PIN subjects: a preliminary report from a 1 year proof of principle study. Proc Am Assoc Cancer Res. 2005;46:[abstract #4400]
- Pharmacokinetics and safety of green tea polyphenols after multiple-dose administration of epigallocatechin gallate and polyphenon E in healthy individuals. Clin Cancer Res. 2003;9:3312–3319
- A single ascending dose study of epigallocatechin gallate in healthy volunteers. J Int Med Res. 2003;31:88–101
- Effects of dosing condition on the oral bioavailability of green tea catechins after single-dose administration of polyphenon E in healthy adults. Proc Am Assoc Cancer Res. 2005;46:B70
- Phase I trial of oral green tea extract in adult patients with solid tumors. J Clin Oncol. 2001;19:1830–1838
- Spontaneous clinical regression in chronic lymphocytic leukaemia. Br J Haematol. 2002;116:341–345
- . Spontaneous remission of b-chronic lymphocytic leukaemia. Br J Haematol. 2002;119:874–875
- . “Spontaneous” complete remissions in chronic lymphocytic leukemia: report of three cases and review of the literature. Blood Cells. 1987;12:471–483
PII: S0145-2126(05)00421-2
doi: 10.1016/j.leukres.2005.10.020
© 2005 Elsevier Ltd. All rights reserved.
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Leukemia Research
Volume 30, Issue 6
, Pages 707-712
, June 2006
