Next »
Leukemia Research
Volume 25, Issue 12
, Pages 1033-1045
, December 2001
Detailed clonality analysis of relapsing precursor B acute lymphoblastic leukemia: implications for minimal residual disease detection
References
- . Detection of T-cell receptor gamma chain V gene rearrangements using the polymerase chain reaction: application to the study of clonal disease cells in acute lymphoblastic leukemia. Blood. 1991;77:1989–1995
- . The significance of detection of minimal residual disease in childhood acute lymphoblastic leukaemia. Br. J. Haematol. 1993;83:412–418
- A polymerase chain reaction study of the stability of Ig heavy-chain and T-cell receptor delta gene rearrangements between presentation and relapse of childhood B-lineage acute lymphoblastic leukemia. Blood. 1994;83:1355–1362
- Clonal rearrangements in childhood and adult precursor B acute lymphoblastic leukemia: a comparative polymerase chain reaction study using multiple sets of primers. Eur. J. Haematol. 1999;63:211–218
- . Detection of minimal residual disease in acute leukemia: methodologic advances and clinical significance. Blood. 1995;85:1416–1434 (see comments)
- . Detection of minimal residual disease in acute leukemia by flow cytometry. Cytometry. 1999;38:139–152
- Detection of minimal residual disease in acute leukemia patients. Cytokines Mol. Ther. 1996;2:121–133
- Prognostic value of minimal residual disease in acute lymphoblastic leukaemia in childhood. Lancet. 1998;352:1731–1738 (see comments)
- Standardized RT-PCR analysis of fusion gene transcripts from chromosome aberrations in acute leukemia for detection of minimal residual disease. Report of the BIOMED-1 Concerted Action: investigation of minimal residual disease in acute leukemia. Leukemia. 1999;13:1901–1928
-
Clinical significance of minimal residual disease in childhood acute lymphoblastic leukemia. European Organization for Research and Treatment of Cancer — Childhood Leukemia Cooperative Group.
New Engl. J. Med. 1998;339:591–598
(see comments)
- . Monitoring residual disease in acute lymphoblastic leukemia: therapeutic implications. Cytokines Mol. Ther. 1995;1:65–69
-
.
Rapid molecular response during early induction chemotherapy predicts a good outcome in childhood acute lymphoblastic leukemia.
Blood. ;95. 2000;95:790–794
(published erratum appeared in Blood May 15;95(10):3010)
- Immunological detection of minimal residual disease in children with acute lymphoblastic leukaemia. Lancet. 1998;351:550–554 (see comments)
- Prospective monitoring and quantitation of residual blasts in childhood acute lymphoblastic leukemia by polymerase chain reaction study of delta and gamma T-cell receptor genes. Blood. 1994;83:1892–1902
- . Detection of minimal residual disease in all: biology, methods, and applications. Cancer Treat. Res. 1996;84:149–166
- Analysis of junctional diversity in the preferential V delta 1-J delta 1 rearrangement of fresh T-acute lymphoblastic leukemia cells by in vitro gene amplification and direct sequencing. Blood. 1989;74:2053–2061
- . Use of oligonucleotide probes directed against T cell antigen receptor gamma delta variable-(diversity)-joining junctional sequences as a general method for detecting minimal residual disease in acute lymphoblastic leukemias. J. Clin. Invest. 1990;86:2125–2135
- . Analysis of Ig and T-cell receptor genes in 40 childhood acute lymphoblastic leukemias at diagnosis and subsequent relapse: implications for the detection of minimal residual disease by polymerase chain reaction analysis. Blood. 1994;83:2238–2247
- Characterization of clonal immunoglobulin heavy chain and T cell receptor gamma gene rearrangements during progression of childhood acute lymphoblastic leukemia. Leukemia. 1995;9:1847–1850
- Clonal evolution as judged by immunoglobulin heavy chain gene rearrangements in relapsing precursor-B acute lymphoblastic leukemia. Eur. J. Haematol. 1999;63:171–179
- . Immunoglobulin heavy chain gene VH–D junctional diversity at diagnosis in patients with acute lymphoblastic leukemia. Blood. 1993;81:775–782
- . Distinct ongoing Ig heavy chain rearrangement processes in childhood B-precursor acute lymphoblastic leukemia. Blood. 1993;82:581–589
- . Frequent ongoing T-cell receptor rearrangements in childhood B-precursor acute lymphoblastic leukemia: implications for monitoring minimal residual disease. Blood. 1995;86:692–702
- Clonal evolution in B-lineage acute lymphoblastic leukemia by contemporaneous VH–VH gene replacements and VH–DJH gene rearrangements. Blood. 1996;87:2506–2512
- . Lymphocytic progenitor cell origin and clonal evolution of human B-lineage acute lymphoblastic leukemia. Blood. 1996;88:609–621
- . Detection of clonal B cell populations in paraffin-embedded tissues by polymerase chain reaction. Am. J. Pathol. 1993;143:230–239
- . Detection of clonal lambda light chain gene rearrangements in frozen and paraffin-embedded tissues by polymerase chain reaction. Am. J. Pathol. 1995;147:806–814
- VH gene rearrangement events can modify the immunoglobulin heavy chain during progression of B-lineage acute lymphoblastic leukemia. Blood. 1992;79:223–228 (see comments)
- . Immunoglobulin heavy chain gene fingerprinting reveals widespread oligoclonality in B-lineage acute lymphoblastic leukaemia. Leukemia. 1991;5:832–838
- . Simultaneous immunoglobulin/T-cell receptor gene rearrangements and multiclonality in childhood acute lymphoblastic leukemia. Acta Paediatr. 1994;83:319–326
- . Recombination between an expressed immunoglobulin heavy-chain gene and a germline variable gene segment in a Ly 1+ B-cell lymphoma. Nature. 1986;322:843–846
- . A novel VH to VHDJH joining mechanism in heavy-chain-negative (null) pre-B cells results in heavy-chain production. Nature. 1986;322:840–842
- Late relapsing childhood lymphoblastic leukemia. Blood. 1998;92:2334–2337
- Clonal stability in children with acute lymphoblastic leukemia (ALL) who relapsed five or more years after diagnosis. Leukemia. 1999;13:190–195
- . Oligoclonal immunoglobulin heavy-chain and T-cell receptor delta rearrangements persist in a recurrent acute lymphoblastic leukemia with one immunoglobulin kappa rearrangement as a clonal marker. Mod. Pathol. 1999;12:819–826
-
.
Light-chain gene expression before heavy-chain gene rearrangement in pre-B cells transformed by Epstein-Barr virus.
Proc. Natl. Acad. Sci. USA. 1989;86:2356–2360
- Multiple rearranged immunoglobulin genes in childhood acute lymphoblastic leukemia of precursor B-cell origin. Leukemia. 1991;5:657–667
- Clonal diversity of Ig and T-cell-receptor gene rearrangements identifies a subset of childhood B-precursor acute lymphoblastic leukemia with increased risk of relapse. Blood. 1998;92:952–958
PII: S0145-2126(01)00072-8
© 2001 Elsevier Science Ltd. All rights reserved.
Next »
Leukemia Research
Volume 25, Issue 12
, Pages 1033-1045
, December 2001
